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Developmental Psychopathology, moment version, comprises in 3 volumes the main whole and present study on each point of developmental psychopathology. This seminal reference paintings beneficial properties contributions from nationwide and overseas professional researchers and clinicians who collect an array of interdisciplinary paintings to envision how a number of degrees of research might impact person ameliorations, the continuity or discontinuity of styles and the pathways in which an identical developmental results will be completed. This quantity addresses theoretical views and methodological concerns, together with cross-cultural views, developmental epidemiology, self selection concept, and gender concerns.
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Extra info for Developmental Psychopathology, Theory and Method . Volume 1.(WILEY SERIES ON PERSONALITY PROCESSES)
Schizotaxia) postulated to exist in Schizophrenia by Paul Meehl (1962). Importantly, Fish noted that the phenotypic manifestations of the neurointegrative defect change over epigenesis and that the signs of dysregulation of maturation are found in many developing systems. Specifically, Fish reported that the neurointegrative disorder present from infancy disrupted the normal timing, sequence, and overall organization of development. Moreover, in a landmark prospective longitudinal investigation, Fish, Marcus, Hans, Auerbach, and Perdue (1992) discovered that the infant offspring of schizophrenic mothers displayed greater lags in their motor development during infancy and that a number of these infants themselves went on to develop Schizophrenia or schizotypal personality disorders (see also the seminal work of Walker, Davis, & Gottlieb, 1991, in this regard).
Introducing birth complications and teratogens may also place the cortical connections being established and refined at increased risk for aberrant development. 2. A number of postmortem neuropathology studies has found evidence of heterotopic displacement of neurons in various regions of the brain, including the hippocampus and the frontal and temporal cortices. These findings suggest that there are disturbances of brain development in utero in many schizophrenics. 3. Disturbances in neurogenesis, neuronal migration and differentiation, synaptogenesis, neuronal and synaptic pruning (perhaps resulting in reduced synaptic connectivity; cf.
These multiple pathways embrace a number of possible contributors that may potentiate or mediate the links between early neurodevelopmental anomalies and Schizophrenia in genetically vulnerable individuals. These include the normal developmental changes that take place during late adolescence and early adulthood, such as: (1) synaptic pruning of the prefrontal cortex (Feinberg, 1982; McGlashan & Hoffman, 2000), (2) pubertal increases in gonadal hormones during adolescence (Spear, 2000; Walker, Sabuwalla, & Huot, 2004), (3) developmental transformations in prefrontal cortex and limbic brain regions (Dahl, 2004; Keshavan & Hogarty, 1999; Marenco & Weinberger, 2000), (4) continued myelination of intracortical connections (Benes, 1989; Gibson, 1991; Yakovlev & LeCours, 1967), (5) alterations in the balance between mesocortical and mesolimbic dopamine systems (Benes, 1989, 1997; Benes, Turtle, Khan, & Farol, 1994), (6) the stress that arises during postnatal social development (Keshavan & Hogarty, 1999; Walker & Diforio, 1997), (7) the transformations that occur in cognitive and social-cognitive development (Keating, 1990; Noam, Chandler, & LaLonde, 1995; Spear, 2000), and (8) the growing importance of the peer group (J.