By W. E. Johns

This comprises sixteen brief tales all of that are strange in that they encompass Ginger telling his comrades in 666 (Fighter) Squadron of his adventures with Biggles once they have been chartered by means of Dr. Augustus Duck (nicknamed "Donald") to take him to enquire a variety of unusual phenomena.

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Instead the low risk breast cancer group excreted decreased levels of estrone and l7S-estradiol. g. estriol) "protects" target tissues from the carcinogenic (77,81) effect of the more active estrogens. Since pregnancy results in very high levels of urinary estriol, this viewpoint has been extrapolated to explain the protecting effect of early first pregnancy on the incidence of breast *During the preparation of this manuscript an excellent review entitled "Hormone Profiles in Hormone-Dependent Cancers" by B.

In experimental animals hepatic mixed oxidases (cytochrome P450) are known to be involved in steroid hydroxlations (52). These microsomal enzymes are responsible for the hydroxylation of steroids at positions 2,68, 78, l5a, l6a, l7a and 21 (28-33,53,54). Other substrates such as fatty acids, drugs, anesthetics, insecticides, and carcinogens are also hydroxylated or OXidatively N-demethylated by this system in the presence of NADPH and oxygen. Both androgens and corticosteroids have been shown to activate this hepatic microsomal mono-oxygenase system although through different mechanisms (55).

Urine specimens were collected from some 5,000 healthy women. When breast cancer was subsequently discovered in a woman, her prediagnosis excretion of l7-ketosteroids and l7ahydroxycorticosteroids was compared with matched controls. At the time of publication 27 women had presented with breast cancer. The mean excretion of etiocholanolone in the precancer cases was significantly lower than that of the matched controls. This abnormality in l7-ketosteroids excretion was found at all ages and was seen as early as nine years prior to the diagnosis of breast cancer.

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